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  • Hi! It's Me!
  • Advising
  • Blog
  • My Old Blog
    • Home (Read Me First) >
      • High School
      • College Admissions
      • College
      • Other Stuff
      • Free Editing
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YOUR CART

Tanvi Banota

Scientist. Writer. Fencer. Mentor. ...(I've run out of good nouns.)

Get to know me better:

More About Me
My Research
My Blog
My CV
 

More About Me

I am an MD/PhD student at the Yale School of Medicine, planning on pursuing a PhD in Immunology and becoming a physician-scientist leader in academic medicine.

I graduated from Rutgers University - Honors College in 2022, with a degree in Cell Biology and Neuroscience and minor in Linguistics.

If you don't find me in the lab, you'll find me ruminating about life (more on this on my blog), stabbing people with my epee (go Rutgers Fencing!), making lettering art pieces, learning a new language, or jamming out on my piano and ukulele. 
 

My Research

I work in Dr. Debra Laskin's laboratory in the Department of Pharmacology and Toxicology at Rutgers University. I first joined the lab as a high schooler as part of the ​Liberty Science Center Partners in Science Program in 2017 and have been working there since! The Laskin Lab focuses on investigating the macrophage-mediated inflammatory response in toxicant-induced pulmonary injury. I was named a 2020 Goldwater Scholar for my research achievements. Read more about my projects below:
Regulation of Macrophage Phenotype by
Farnesoid X Receptor during
Nitrogen Mustard-Induced Lung Injury
Picture
Source: Laskin 2009 "Battle of Forces"
Using nitrogen mustard to induce pulmonary injury and inflammation yields a tightly regulated, balanced macrophage response. We are investigating the role of FXR, a gene that may play a role in modulating this inflammatory macrophage response.
 Development of Pulmonary Inflammation
and Injury in a Mouse Model of
Non-Alcoholic Steatohepatitis (NASH)
Picture
Source: Ota 2016 "NAFLD Macrophage Polarization"
Non-alcoholic steatohepatisis (NASH) is a chronic liver injury characterized by fat accumulation and inflammation. Using a NASH injury model, we are investigating the development of pulmonary inflammation in response to NASH, to elucidate the possible pathways involved in lung-liver cross talk.

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